Base de dados : HANSEN Pesquisa : CELULAS DE SCHWANN [Descritor de assunto] Referências encontradas : 34 [refinar] Mostrando: 1 .. 20   no formato [Detalhado]

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Id: 25109 Autor: Alves, Lucineia; Lima , Leila de Mendonca; Maeda, Elisa da Silva; Carvalho, Lais; Holy, Jon; Sarno, Euzenir Nunes; Pessolani, Maria Cristina Vidal; Barker, Lucia P Título: Mycobacterium lepre infection of human Schwann cells depends on selective host kinases and pathogen-modulated endocytic pathways ..- Fonte: s.l; s.n; 2004. 9 p. ilus, graf. Resumo: Mycobacterium leprae, an obligate intracellular pathogen, shows a unique tropism for Schwann cells (SC). This leads to the peripheral neuropathy disorde observed in leprosy. In this study, we investigated signal transduction events and the intracellular fate of M. leprae during the interaction of the microorganism with SC. First, we demonstrated that the human schannoma cell line ST88-14 readily phagocytized the bacteria as observed by time-lapse microscopy, actin staining and electron microscopy. The effect of specific kinase inhibitors on M. leprae internalization was then investigated showing that functional protein tyrosine kinase, calcium-dependent protein kinase and phosphatidylinositol 3-kinase, but not cAMP-dependent protein kinase are essential for phagocytosis of the bacteria. Similar results were obtained when irradiated and live bacteria were compared and when M. leprae was pre-coated with recombinant histone-like protein/laminin binding protein, a bacterial adhesion. In a addition, experiments were perfomed to analyze the bacterial trafficking within the endosomal network by labeling the acidified intracellular compartments of M. leprae-infected SC with the Lysotracker acidotrophic probe. Acidification of vesicles containing live M. leprae was minimal in both RAW murine macrophages and SC, although phagosomes containing heat-killed bacteria seem to follow normal endocytic maturation. These data indicate that the invading bacteria interfere with normal endocytic pathway maturation of bacteria-containing phagosomes within SC (AU). Descritores: MYCOBACTERIUM LEPRAE/imunol MYCOBACTERIUM LEPRAE/patogen CELULAS DE SCHWANN/microbiol - HANSENIASE/imunol  HANSENIASE/microbiol Limites: HUMANO ANIMAL Localização: BR191.1. 9199/s

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Id: 18643 Autor: Hagge, Deanna A; Robinson, Sandra Oby; Scollard, David; McCormick, Gregory; Williams, Diana L Título: A new model for studyng the effects of Mycobacterium leprae on Schwann cell and neuron interactions ..- Fonte: s.l; s.n; 2002. 14 p. ilus, tab. Resumo: Millions of patients with leprosy suffer from nerve damage resulting in disabilities as a consequence of Mycobacterium leprae infection. However, mechanisms of nerve damage have not been elucidated because of the lack of a model that maintains M. leprae viability and mimics disease conditions. A model was developed using viable M. leprae, rat Schwann cells, and Schwann cell-neuron cocultures incubated at 33 degrees C. M. leprae retained 56 per cente viability in Schwann cells for 3 weeks after infection at 33 degrees C, compared with 3.6 per cente viability at 37 degrees C. Infected Schwann cells had altered morphology and expression of genes encoding cellular adhesion molecules at 33 degrees C but were capable of interacting with and myelinating neurons. Cocultures, infected after myelination occurred, showed no morphological changes in myelin architecture after 1 month of incubation at 33 degrees C, and M. leprae retained 53 per cente viability. This article describes a new model for studying the effects of M. leprae on Schwann cells. (AU). Descritores: CAMUNDONGOS NUS HANSENIASE/microbiol HANSENIASE/patol MODELOS NEUROLOGICOS MICROSCOPIA ELETRÔNICA DE VARREDURA CELULAS CULTIVADAS NEURÔNIOS/microbiol NEURÔNIOS/patol NEURÔNIOS/fisiol COMUNICACAO CELULAR/fisiol MYCOBACTERIUM LEPRAE/cresc MYCOBACTERIUM LEPRAE/fisiol CELULAS DE SCHWANN/microbiol CELULAS DE SCHWANN/patol CELULAS DE SCHWANN/fisiol CELULAS DE SCHWANN/ultraest Limites: HUMANO ANIMAL CAMUNDONGOS RATOS SUPPORT, NON-U.S. GOV'T Meio Eletrônico: - . Localização: BR191.1; 09030/s

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Id: 18580 Autor: Oliveira, Rosane B; Ochoa, Maria T; Sieling, Peter A; Rea, Thomas H; Rambukkana, Anura; Sarno, Euzenir N; Modlin, Robert L Título: Expression of toll-like receptor 2 on human Schwann cells: a mechanism of nerve damage in leprosy ..- Fonte: s.l; s.n; 2003. 7 p. ilus, graf. Resumo: Nerve damage is a clinical hallmark of leprosy and a major source of patient morbidity. We investigated the possibility that human Schwann cells are susceptible to cell death through the activation of Toll-like receptor 2 (TLR2), a pattern recognition receptor of the innate immune system. TLR2 was detected on the surface of human Schwann cell line ST88-14 and on cultured primary human Schwann cells. Activation of the human Schwann cell line and primary human Schwann cell cultures with a TLR2 agonist, a synthetic lipopeptide comprising the N-terminal portion of the putative Mycobacterium leprae 19-kDa lipoprotein, triggered an increase in the number of apoptotic cells. The lipopeptide-induced apoptosis of Schwann cells could be blocked by an anti-TLR2 monoclonal antibody. Schwann cells in skin lesions from leprosy patients were found to express TLR2. It was possible to identify in the lesions Schwann cells that had undergone apoptosis in vivo. The ability of M. leprae ligands to induce the apoptosis of Schwann cells through TLR2 provides a mechanism by which activation of the innate immune response contributes to nerve injury in leprosy. (AU). Descritores: APOPTOSE CELULAS CULTIVADAS DANO DO DNA IMUNIDADE NATURAL HANSENIASE/imunol HANSENIASE/patol LIPOPROTEINAS/fisiol CELULAS DE SCHWANN/patol GLICOPROTEINAS DE MEMBRANA/anal GLICOPROTEINAS DE MEMBRANA/fisiol RECEPTORES DA SUPERFICIE CELULAR/anal RECEPTORES DA SUPERFICIE CELULAR/fisiol Limites: HUMANO SUPPORT, NON-U.S. GOV'T SUPPORT, U.S. GOV'T, P.H.S Meio Eletrônico: - . Localização: BR191.1; 09028/s

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Id: 18455 Autor: Job, Charles K Título: Mycobacterium leprae in nerve lesions in lepromatous leprosy ..- Fonte: s.l; s.n; mar. 1970. 13 p. ilus. Resumo: The ultrastructure of radial cutaneous nerve biopsies from five untreated patients with lepromatous leprosy was studied. Mycobacterium leprae prenet in large numbers frequently in Schwann cells, macrophages, and endothelial cells, and occasionally in perineural cells. Proliferation of the bacilli inside these cells resulted in their foamy degeneration. It is pointed out that the Schawann cells is the target cell in the leprous infection of the nerves and that the destructive lesions of the nerves produced in lepromatous leprosy are esentially the result of Schwann-cell degeneration. Mycobacterium leprae was demonstrated in the lumen of the capillaries as well, and it is suggested that the infection may spread in the body even into the nerves through the blood-stream.(AU). Descritores: HANSENIASE/microbiol HANSENIASE/patol MICROSCOPIA ELETRÔNICA MYCOBACTERIUM LEPRAE/isol CELULAS DE SCHWANN Limites: HUMANO Meio Eletrônico: - . Localização: BR191.1; 00513/s

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Id: 18340 Autor: Hagge, Deanna; Robinson, Sandra Oby; Scollard, David; McCormick, Gregory; Williams, Diana L Título: A new model for studying the effects of Mycobacterium leprae on Schwann cell and neuron interactions ..- Fonte: s.l; s.n; 2002. 4 p. tab. Resumo: Millions of patients with leprosy suffer from nerve damage resulting in disabilities as a consequence of Mycobacterium leprae infection. However, mechanisms of nerve damage have not been elucidated because of the lack of a model that maintains M. leprae viability and mimics disease conditions. A model was developed using viable M. leprae, rat Schwann cells, and Schwann cell-neuron cocultures incubated at 33 degrees C. M. leprae retained 56% viability in Schwann cells for 3 weeks after infection at 33 degrees C, compared with 3.6% viability at 37 degrees C. Infected Schwann cells had altered morphology and expression of genes encoding cellular adhesion molecules at 33 degrees C but were capable of interacting with and myelinating neurons. Cocultures, infected after myelination occurred, showed no morphological changes in myelin architecture after 1 month of incubation at 33 degrees C, and M. leprae retained 53% viability. This article describes a new model for studying the effects of M. leprae on Schwann cells. (AU). Descritores: COMUNICACAO CELULAR/fisiol CELULAS CULTIVADAS HANSENIASE/microbiol HANSENIASE/patol CAMUNDONGOS NUS MODELOS NEUROLOGICOS NEURÔNIOS/microbiol NEURÔNIOS/patol NEURÔNIOS/fisiol CELULAS DE SCHWANN/microbiol CELULAS DE SCHWANN/patol CELULAS DE SCHWANN/fisiol CELULAS DE SCHWANN/ultraest MICROSCOPIA ELETRÔNICA DE VARREDURA MYCOBACTERIUM LEPRAE/cresc MYCOBACTERIUM LEPRAE/fisiol Limites: ANIMAL HUMANO CAMUNDONGOS RATOS SUPPORT, NON-U.S. GOV'T Localização: BR191.1; 08982/s

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Id: 18131 Autor: Ridley, D. S Título: The pathogenesis of the early skin lesion in leprosy ..- Fonte: s.l; s.n; nov. 1973. 16 p. ilus, tab. Resumo: A study of early skin lesions of leprosy patients, many the with a history known to be of less than 1 years duration, showed that by the use of serial sections the great majority could be classified on an immunological basis.(AU). Descritores: CELULAS DE SCHWANN HANSENIASE/clas HANSENIASE/imunol HANSENIASE/microbiol HANSENIASE/patol NEURÔNIOS/microbiol PELE/irrig PELE/inerv PELE/microbiol PELE/patol Limites: HUMANO Meio Eletrônico: - . Localização: BR191.1; 00029/s

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Id: 18026 Autor: Anthony, J; Vaidya, M. C; Dasgupta, A Título: Ultrastructure of nerve in erythema nodosum leprosum (ENL) ..- Fonte: s.l; s.n; 1979. 4 p. ilus. Resumo: Gross changes of myelinated fibres and Schwann cells at different degenerative stages were present in all five ENL nerve lesions. Besides these changes, infiltrating cells mainly macrophages, deposition of excessive collagen and perineurial vessel damage were also observed.(AU). Descritores: CELULAS DE SCHWANN/ultraest ERITEMA NODOSO/patol HANSENIASE/patol DEGENERACAO NEURAL FIBRAS NERVOSAS MIELINIZADAS/ultraest - SISTEMA NERVOSO/ultraest  COLAGENO  ORELHA/inerv  MICROSCOPIA ELETRÔNICA  CELULAS SANGUINEAS Limites: HUMANO Meio Eletrônico: - . Localização: BR191.1; 01060/s

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Id: 17933 Autor: Sunderland, Sidney Título: The internal anatomy of nerve trunks in relation to the neural lesions of leprosy ..- Fonte: s.l; s.n; dec. 1973. 23 p. ilus, tab. Resumo: The internal anatoly of peripheral nerve trunks has been studied and discussed in relation to some features of the neural lesions of leprosy. Special significance is attached to the perineurium, funicular structure and the intraneural funicular plexuses.The perineurium controls the spread of the micro-organisms confining them, for some time, to the affected funiculi and promoting their movement centrally along the bundles. By the time the outlines of the perineurium have been lost and the epineurium involved at any level, the micro-organisms are likley to have advanced considerable distances up the bundles.Because of the elastic resistance of the perineurium, the pressure inside the bundle builds up as the products of the inflammatory reaction slowly accumulate. Compression and ischemia of the contents of the bundle introduced in this way add to the involvement and destruction of nerve fibres due directly to the activity of the M. leprae.(AU). Descritores: TECIDO CONJUNTIVO/patol ISQUEMIA HANSENIASE/compl HANSENIASE/imunol HANSENIASE/microbiol HANSENIASE/patol HANSENIASE/fisiopatol HANSENIASE/cirurg MYCOBACTERIUM LEPRAE/patogen CELULAS DE SCHWANN - PRESSAO  SENSACAO Meio Eletrônico: - . Localização: BR191.1; 00529/s

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Id: 17808 Autor: Job, C. K Título: Mechanism of nerve destruction in tuberculoid-borderline leprosy ..- Fonte: s.l; s.n; sep. 1973. 14 p. ilus. Resumo: The ultrastructural changes in radial cutaneous nerve biopsies from 3 tuberculoid and 14 tuberculoid-borderline patients were studied. In tuberculoid leprosy the nerve parenchyma was almost entirely replaced by tuberculoid granuloma. In nerves from borderline-tuberculoid leprosy many axons showed segmental demyelination and Wallerian degeneration. The nerve tissue was gradually replaced by proliferating collagen fibrils and inflammatory granulomata. M. leprae were rare and when present they were found in phagolysosomes of Schwann cells and macrophages. Schwann cells were surrounded and occasionally infiltrated by lymphocytes and macrophages. It was suggested that "sensitized Schwann cells" were the target of a hypersensivity reaction resulting in their degeneration or death which in turn produced segmental demyelination or Wallerian degeneration.(AU). Descritores: HANSENIASE/imunol HANSENIASE/microbiol HANSENIASE/patol DOENCAS DESMIELINIZANTES ANTIGENOS DE BACTERIAS MICROSCOPIA ELETRÔNICA MYCOBACTERIUM LEPRAE/isol NERVOS PERIFERICOS/patol CELULAS DE SCHWANN Limites: HUMANO Meio Eletrônico: - . Localização: BR191.1; 02021/s

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Id: 17791 Autor: Dayan, A. D; Sandbank, U Título: Pathology of ther peripheral nerves in leprosy: report of a case ..- Fonte: s.l; s.n; oct. 1970. 6 p. ilus, graf. Resumo: Peripheral nerve trunks were examined from a case of lepromatous leprosy. M. leprae was seen in the nerves in several types of cells. isolated nerve fibre preparations showed demyelinating lesions due to Schwann cell damage and complete nerve fibre degeneration. The causes of this mixed pattern of damage may involve both a direct effect of the bacilli on nerve fibres and various epiphenomena.(AU). Descritores: DOENCAS DESMIELINIZANTES/patol HANSENIASE/patol NERVO FEMORAL/patol AUTOPSIA NERVOS PERIFERICOS/patol CELULAS DE SCHWANN NERVO CIATICO/patol DEGENERACAO NEURAL NERVO ULNAR/patol REGENERACAO NERVOSA Limites: HUMANO IDOSO MASCULINO Meio Eletrônico: - . Localização: BR191.1; 02018/s

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Id: 17780 Autor: Shetty, V. P; Mehta, L. N; Irani, P. F; Antia, N. H Título: Study of the evolution of nerve damage in leprosy. Part I--Lesions of the index branch of the radial cutaneous nerve in early leprosyStudy of evolution of nerve damage in leprosy. Part II--Observations on the index branch of the radial cutaneous nerve in contacts of leprosy ..- Fonte: s.l; s.n; jan. 1980. 14 p. tab, graf, ilus. Resumo: A total of six index finger branch of the radial cutaneous (IRC) nerves from three BL, two BB and one BT cases of leprosy, with less than 6 months history of disease were subjected for nerve conduction velocity (NCV) studies followed by biopsy. The biopsy was divided into three parts and subjected for light microscopy quantitative histology, electron-microscopy and fibre tease studies. The nerves revealed a slight reduction in NCV and a varying degree of segmental demyelination ranging from 10% to 35%. The earliest changes observed were the thickening and proliferation of Schwann cell processes of the unmyelinated fibres and degeneration of their axons regardless of the type of leprosy.(AU) A total of 35 contacts from 20 families of lepromatous index cases were screened clinically. All of them were subjected for nerve conduction velocity (NCV) studies of both left and right index branch of the radial cutaneous (IRC) nerve. Fifteen of these nerves were biopsied. Out of 15 IRC nerves biopsied 10 had slightly reduced average NCV values and 5 were normal. Four nerves out of 10 where average NCV was reduced and 1 nerve out of 5 where NCV was normal showed significant ultrastructural changes and higher percentage of segmental demyelination (10%--13%). There was a striking similarity between the changes seen in the IRC nerves of early cases of leprosy and of contacts.(AU). Descritores: PELE/irrig FATORES DE TEMPO HANSENIASE/patol DEGENERACAO NEURAL FIBRAS NERVOSAS/ultraest CONDUCAO NERVOSA NERVO RADIAL/patol NERVO RADIAL/ultraest DEDOS/irrig HANSENIASE/diag HANSENIASE/genet CELULAS DE SCHWANN/ultraest Limites: MASCULINO FEMININO ADOLESCENTE ADULTO CRIANCA HUMANO MEIA-IDADE Meio Eletrônico: - . Localização: BR191.1; 01984/s

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Id: 16405 Autor: Brophy, Peter J Título: Subversion of Schwann cells and the leper's bell ..- Fonte: s.l; s.n; May 2002. 2 p. ilus. Descritores: AXONIOS MORTE CELULAR DIFERENCIAÇAO CELULAR MEMBRANA CELULAR COCULTURA PROTEINAS CITOESQUELETO DOENÇAS DESMIELINIZANTES DISTROFINA GLICOLIPIDIOS LAMININA HANSENIASE HANSENIASE HANSENIASE GLICOPROTEINAS DE MEMBRANAS PROTEINAS DA MEMBRANA CAMUNDONGOS MYCOBACTERIUM LEPRAE MYCOBACTERIUM LEPRAE MYCOBACTERIUM LEPRAE BAINHA DE MIELINA CÉLULAS DE SCHWANN CÉLULAS DE SCHWANN CÉLULAS DE SCHWANN Limites: ANIMAL Localização: BR191.1; 08621/s

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Id: 16382 Autor: Sarno, Euzenir Nunes; Pessolani, Maria Cristina Vidal Título: Oldest and most feared disease ..- Fonte: s.l; s.n; Dec. 2000. 1 p. ilus. Descritores: DOENÇAS DESMIELINIZANTES HANSENIASE HANSENIASE HANSENIASE MYCOBACTERIUM LEPRAE CÉLULAS DE SCHWANN CÉLULAS DE SCHWANN FATOR DE NECROSE TUMORAL/PH Localização: BR191.1; 08656/s

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Id: 16357 Autor: Gerosa, P. L; Spinelli, M; Giussani, G; Vai, C; Fontana, A; Canepari, C Título: Neurofibromatosi(NF1) e neurolabbra: immunoreazione verso cellule di Schwann patologiche Neurofibromatosis (NF1) and neuroleprosy: immunoreaction against pathologic Schwann-cells physiopathogenetic observations- Fonte: s.l; s.n; 2001. 9 p. ilus, tab. Descritores: IMUNOHISTOQUIMICA HANSENIASE HANSENIASE DIMORFA DOENÇAS DO SISTEMA NERVOSO NEUROFIBROMATOSE CÉLULAS DE SCHWANN Localização: BR191.1; 08695/s

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Id: 16355 Autor: Spierings, Eric; Boer, Tjitske de; Wieles, Brigitte; Adams, Linda B; Marani, Enrico; Ottenhoff, Tom H. M Título: Mycobacterium leprae-specific, HLA class II- restricted killing of human Schwann cells by CD4+ Th1 cells: a novel immunopathogenic mechanism of nerve damage in leprosy ..- Fonte: s.l; s.n; 2001. 6 p. ilus, tab, graf. Descritores: APRESENTAÇAO DO ANTIGENO ANTIGENOS DE BACTÉRIAS ANTIGENOS DE BACTÉRIAS CÉLULAS CLONAIS CITOTOXICIDADE IMUNOLOGICA MAPEAMENTO DE EPITOPOS EPITOPOS DE LINFOCITO T ANTIGENOS HLA-D IMUNOFENOTIPAGEM HANSENIASE HANSENIASE HANSENIASE CAMUNDONGOS MYCOBACTERIUM LEPRAE CÉLULAS DE SCHWANN CÉLULAS DE SCHWANN CÉLULAS DE SCHWANN CÉLULAS TH1 CÉLULAS TH1 CÉLULAS TH1 Localização: BR191.1; 08693/s

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Id: 16337 Autor: Rambukkana, Anura; Zanazzi, George; Tapinos, Nikos; Salzer, James L Título: Contact-dependent demyelination by mycobacterium leprae in the absence of immune cells ..- Fonte: s.l; s.n; May 2002. 5 p. ilus, graf. Descritores: APOPTOSE AXONIOS AXONIOS LINFOCITOS B ADERENCIA BACTERIANA DIVISAO CELULAR COCULTURA DOENÇAS DESMIELINIZANTES GANGLIOS ESPINHAIS GENES RAG-1 GLICOLIPIDIOS HANSENIASE HANSENIASE HANSENIASE HANSENIASE MACROFAGOS CAMUNDONGOS CAMUNDONGOS ENDOGAMICOS C57BL CAMUNDONGOS KNOCKOUT MUTAÇAO MYCOBACTERIUM LEPRAE MYCOBACTERIUM LEPRAE BAINHA DE MIELINA BAINHA DE MIELINA DEGENERAÇAO NEURAL FIBRAS NERVOSAS MIELINIZADAS NEURONIOS CÉLULAS DE SCHWANN CÉLULAS DE SCHWANN NERVO CIATICO NERVO CIATICO LINFOCITOS T CULTURA DE TECIDO Limites: ANIMAL Localização: BR191.1; 08647/s

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Id: 16290 Autor: Young, Douglas B Título: Leprosy lipid provides the key to SChwann cell entry ..- Fonte: s.l; s.n; feb. 2001. 3 p. . Descritores: GLICOLIPIDIOS HANSENIASE MYCOBACTERIUM LEPRAE MYCOBACTERIUM LEPRAE CÉLULAS DE SCHWANN Limites: ANIMAL Localização: BR191.1; 08704/s

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Id: 16217 Autor: Rambukkana, Anura Título: Molecular basis for the peripheral nerve predilection of mycobacterium leprae ..- Fonte: s.l; s.n; 2001. 7 p. ilus. Descritores: ANTIGENOS DE BACTÉRIAS PAREDE CELULAR GLICOLIPIDIOS LAMININA HANSENIASE HANSENIASE GLICOPROTEINAS DE MEMBRANAS MODELOS BIOLOGICOS MYCOBACTERIUM LEPRAE NERVOS PERIFÉRICOS LIGAÇAO PROTÉICA CÉLULAS DE SCHWANN TROPISMO Localização: BR191.1; 08702/s

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Id: 16023 Autor: Lockwood, Diana N. J; Colston, M. J; Khanolkar-Young, S. R Título: The detection of Mycobacterium leprae protein and carbohydrate antigens in skin and nerve from leprosy patients with type 1 (reversal) reactions ..- Fonte: s.l; s.n; 2002. 7 p. ilus, tab. Descritores: HANSENIASE DIMORFA HANSENIASE DIMORFA MYCOBACTERIUM LEPRAE MYCOBACTERIUM LEPRAE ANTICORPOS ANTIBACTÉRIAS ANTICORPOS MONOCLONAIS ANTIGENOS DE BACTÉRIAS BIOPSIA CHAPERONINAS CHAPERONINAS HIPERSENSIBILIDADE TARDIA IMUNOHISTOQUIMICA LIPOPOLISSACARIDIOS LIPOPOLISSACARIDIOS MACROFAGOS NERVOS PERIFÉRICOS NERVOS PERIFÉRICOS CÉLULAS DE SCHWANN PELE PELE Localização: BR191.1; 08600/s

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Id: 15968 Autor: Ottenhoff, Tom H. M Título: Mycobacterium leprae and demyelination ..- Fonte: s.l; s.n; 2002. 2 p. . Descritores: HANSENIASE HANSENIASE HANSENIASE DOENÇAS DESMIELINIZANTES DOENÇAS DESMIELINIZANTES DOENÇAS DESMIELINIZANTES MYCOBACTERIUM LEPRAE CÉLULAS DE SCHWANN Localização: BR191.1; 08572/s

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